Isifo se-Alzheimer

Yintoni isifo se-Alzheimer?

Isifo i-Alzheimer sesinye sezizathu eziqhelekileyo zokukhubazeka kubantu abalupheleyo. Luphazamiseko lwemithambo-luvo oluthi ngokuthe ngcembe lubangele ukuncipha kwezicubu zobuchopho kunye nokudodobala kwe-neuronal kwangethuba. Ikwalolona hlobo luxhaphakileyo lwesifo sengqondo esiyingozi, esibangela ukungasebenzi kakuhle kwenkumbulo, izakhono zentlalo, ukucinga nokuziphatha. Ehlabathini jikelele, ngaphezu kwezigidi ezingama-30 abantu abangaphezu kweminyaka engama-65 ubudala baphathwa sisifo i-Alzheimer's.
Izigulana ezinesifo sika-Alzheimer ekuqaleni zibonisa iimpawu zememori ezimbi njengokungakwazi ukukhumbula izehlo zamva nje. Ngokuqhubela kwesifo, isifo se-Alzheimer sinokubangela ukonakala kakhulu kwememori. Ekugqibeleni, isigulana asizukukwazi ukwenza imisebenzi esisiseko yobomi bemihla ngemihla enjengokunxiba ngokwabo, ukutya, ukukhupha amathumbu abo, njalo njalo.

Yintoni i-etiology yesifo se-Alzheimer's?

Isiseko se-etiology yesifo se-Alzheimer asiqondwa ngokucacileyo. Kodwa, uninzi lweengcali kweli candelo zikholelwa ekubeni ukungasebenzi kakuhle kwiiproteni zobuchopho inoxanduva kuthotho lweziganeko ezibangela ukuba ii-neurons zife kwaye ziphazamise ukusebenza kwengqondo. Uphononongo lubonisa ukuba isifo sika-Alzheimer sineetiology ezininzi, kunye nofuzo, indlela yokuphila, kunye nokusingqongileyo okunegalelo ekuphuhlisweni kwesifo sika-Alzheimer.
Kwiimeko ezinqabileyo, uguquko kwimfuzo lwenza ukuba umntu abe semngciphekweni wokuphuhlisa i-Alzheimer's. Kwiimeko ezinjalo ezibangelwa kuguquko, ukuqala kweempawu kwenzeka kwangoko kwaye inkqubela phambili ikhawuleza ngakumbi.
Ngokuqhelekileyo, isifo siqala kwinxalenye yengqondo apho imemori yenziwa khona. Kodwa eyona nkqubo yesifo iqala kudala ngaphambi kokuba isigulana sibe neempawu. Kwinqanaba elihambele phambili lesi sifo, ingqondo iba kumgangatho omangalisayo. Ngokuyintloko, zimbini iiprotein ezichaphazelekayo kwisifo se-Alzheimer's, Beta-amyloid protein, kunye neTau protein.

Iiplaques

I-Beta-amyloid yiprotein eyakhiweyo ephambili enokuba yityhefu kwii-neurons ukuba zidibana kwingqondo. Amacandelo amaqhekeza e-beta-amyloid anokuphazamisa inkqubo yonxibelelwano phakathi kweeseli. Xa la maqela emanyana ngokusondeleleneyo, kutheni kwakhiwa isakhiwo esikhulu esaziwa njengee-amyloid plaque.

Amacangca

Ukuze usebenze ngokufanelekileyo kwee-neurons, iiprotheni ze-tau zidibene nokuthutha izondlo kunye nezinye izinto ezibalulekileyo ukuxhasa ngaphakathi kwi-neurons. Xa iiproteni ze-tau ziphinda zilungelelaniswe zibe ziingqungquthela ezibizwa ngokuba yi-neurofibrillary tangles, zinokubangela isifo se-Alzheimer's. Ezi tangles zingabangela ukuphazamiseka kokuthuthwa kwezondlo kwi-neurons, okubangela ukufa kwabo.

Imiba yomngcipheko weSifo se-Alzheimer

Zininzi izinto ezinokunyusa umngcipheko wesifo se-Alzheimer, ezidweliswe apha ngezantsi.

ubudala

Iminyaka yobudala yeyona nto ibalulekileyo yomngcipheko wokuphuhliswa kwengqondo, kuquka nesifo se-Alzheimer. Nangona kunjalo, i-Alzheimer's ayilophawu lokuguga kwaye ayisiyonto iqhelekileyo yokufumana.

Genetics

Ukuba ilungu elisondeleyo losapho lwakho sele lifunyenwe line-Alzheimer, umngcipheko we-Alzheimer uphezulu kunabantu ngokubanzi.

Isifo esiphantsi

Izigulana ezizalwe zine-Down syndrome, isifo se-chromosomal, zisengozini yokuhlaselwa sisifo i-Alzheimer zisencinci. Ngokwesiqhelo, bakhulisa i-Alzheimer kwishumi lokuqala okanye lesibini lobomi.

Ukulimala kweBrain Injury

Imbali yokubandezeleka entloko okunokuwonyusa umngcipheko wokuba nesifo i-Alzheimer's. Izifundo zibonise ukuba kukho ukwanda kwezehlo ze-Alzheimer kubantu abanesigameko sokwenzakala kwengqondo ebuhlungu.

Ukusela utywala

Ukusela utywala kunokubangela utshintsho olusisigxina kwingqondo. Uphononongo olukhulu lubonise ukuba ukusetyenziswa kotywala kunxulunyaniswa nesifo sengqondo esiyingozi.

ugwayi

Ukuphazamiseka kokulala, njengokulala, kukwanxulunyaniswa nokwanda kweziganeko ze-Alzheimer's kwizifundo ezikhulu.

yokuPhila

Izinto ezinobungozi kwizifo ze-coronary vascular ezifana nokutyeba, uxinzelelo lwegazi, i-cholesterol ephezulu, ukutshaya, kunye nokutshaya. iswekile ziye zanxulunyaniswa nesifo sika-Alzheimer.

Iimpawu kunye neempawu

Lulwazi oluqhelekileyo ukuba uphawu oluphambili lwesifo sika-Alzheimer kukulahleka kwenkumbulo. Kwinqanaba lokuqala lesi sifo, izigulana zineengxaki zokukhumbula iinkumbulo zamva nje kunye neziganeko. Ngokuqhubela phambili kwesifo, imiba ngememori kunye nokwehla kokuqonda.
Ukurhanelwa kwesifo sengqondo esixhalabisayo kuqala kuvela kubahlobo abasenyongweni okanye kumalungu osapho xa iimpawu ziba mandundu ngokwaneleyo ukuba zibonakale. Utshintsho lwezifo kwizicubu zobuchopho lubonisa ngolu hlobo lulandelayo.

Iingxaki zeNkumbulo

Njengoko ukulahleka kwememori kuya kusiba mandundu ngesifo se-Alzheimer, abantu banemicimbi yokunxibelelana kwemihla ngemihla njengokulibala iincoko, ukubeka izinto gwenxa rhoqo, ukulahleka kwiindawo eziqhelekileyo, kunye nokuba nemicimbi yokuqamba izinto okanye intetho yokucinga.

Utshintsho Kubuntu

Isifo i-Alzheimer sinokubutshintsha kakhulu ubuntu nokuziphatha komntu. Ubuntu obuchwayitileyo ngaphambili bunokutshintsha bube kukudakumba koxinzelelo ngelixa kubonisa ukungabikho komdla, ukutshintsha kweemvakalelo kunye nokurhoxa ekuhlaleni.

Kunzima ukwenza izigqibo

Izigulana ezine-Alzheimer's zinobunzima ekwenzeni izigqibo ezifanelekileyo kunye nezigqibo. Umzekelo, isigulana sinokuziphatha ngaphandle komlinganiso kwimikhwa yentlalo enjengokuhamba emvuleni okanye ukuhleka ngexesha lomngcwabo.

Ubunzima ngemisebenzi eqhelekileyo

I-Alzheimer inokuphazamisa amandla omntu okwenza izinto eziqhelekileyo njengokupheka, ukuqhuba, ukudlala imidlalo, njalo njalo. Njengokuba esi sifo siqhubeka, isigulana singaphulukana namandla okwenza imisebenzi yemihla ngemihla enjengokuzinxiba kunye nokungahoyi impilo yabo.

Iingxaki ngokuqiqa

Iingcinga ezingabonakaliyo kunye neengqiqo zinzima kakhulu kubantu abanesifo se-Alzheimer ngenxa yeengxaki zokugxila. Izigulana zinokuba nobunzima ekwenzeni imisebenzi emininzi ngaxeshanye. Imisebenzi yemihla ngemihla ebalulekileyo ekuphileni efana nolawulo lwemali inokuba yinto engenakwenzeka kwizigulana ezine-Alzheimer's.

Sichongwa Njani Isifo i-Alzheimer's?

Uninzi lwezigulana luyaziswa malunga neempawu zazo ngumhlobo osenyongweni okanye ilungu losapho, emva koko isigulana sihlala sifuna unyango. Olunye uvavanyo kufuneka lwenziwe ukuqinisekisa isifo i-Alzheimer's. Olu vavanyo lunokubandakanya ukuvavanywa kwememori yesigulana kunye nezakhono zokuqonda, kunye nezinye iimvavanyo zokucinga. Ukulinganisa kunye nokuvavanywa kwelabhoratri kubalulekile ukuze kuthintelwe ukuxilongwa okwahlukileyo kwe-Alzheimer's. Nangona kunjalo, ukuxilongwa okuqinisekisiweyo kwe-Alzheimer kuhlala kuphela emva kokubhubha kwesigulana njengoko uvavanyo lwe-histopathological yeethishu zobuchopho lubonisa utshintsho olunje nge-neurofibrillary tangles kunye ne-amyloid plaque.

• Uvavanyo lomzimba: Ukuze ulawule ezinye izizathu ezinokubangela isifo sengqondo esixhalabisayo, ugqirha uya kuvavanya ukucinga kwakho, amandla, amandla emisipha kunye neethoni, imisebenzi ye-cranial nerve, ibhalansi kunye nokulungelelanisa.
• Uphando lweelebhu: Ngelixa uvavanyo lwegazi alunakuqinisekisa ukuxilongwa kwe-Alzheimer's, zibalulekile ekuthinteleni usulelo, amathumba, okanye ukusilela kweevithamini, konke oko kunokubangela iimpawu ezifanayo njenge-Alzheimer's. Kwezinye iimeko ezingaqhelekanga, uvavanyo lwe-cerebrospinal fluid lunokwenziwa.
• Uvavanyo lwe-Neurological: Uvavanyo lwenqanaba lengqondo lubandakanya uvavanyo lwezakhono zokuqiqa, inkumbulo kunye nokuqonda. Uvavanyo luthelekisa ukukwazi ukwenza imisebenzi elula yokuqonda kunye nememori kunye nabanye abantu abaneminyaka efanayo ngaphandle kweemeko ze-pathological.
• Ukulinganisa izifundo: Ukuskena ubuchopho nge-MRI okanye i-CT kubalulekile ekwenzeni isifo se-Alzheimer's. Ezi zifundo zokuzicingela zinokunceda ekuchongeni ezinye izizathu zokutshintsha kwemeko yengqondo enje ngokubetha kweschemic, ukopha, amathumba, okanye ukwenzakala. Ukuncipha kwengqondo kunye neendawo zokungasebenzi kakuhle kwemetabolism kunokubonwa kwizifundo zokucinga. Iindlela zokucinga ezintsha ezisebenzisa i-PET scan, i-amyloid PET imaging, kunye ne-Tau PET imaging nayo isaphandwa ngendima yabo ekuchongeni i-Alzheimer's.
• I-Plasma Aβ: I-Plasma Aβ luvavanyo lwegazi olusetyenziselwa ukomeleza ngakumbi ukuxilongwa kwe-Alzheimer's. Luvavanyo olusanda kuqinisekiswa e-US kwaye luyafumaneka ngoku.
• Uvavanyo lweGenetic: Nangona uvavanyo lwemfuzo lungangeni phantsi kovavanyo lwesiqhelo lwe-Alzheimer's, abo banezihlobo zokuqala eziphethwe yi-Alzheimer's banokungena kuvavanyo lwemfuzo.

Ziziphi iingxaki ze-Alzheimer's?

Iingxaki ezinxulumene ne-Alzheimer's ziyafana nenkcazo yeklinikhi. Imiba yenkumbulo, ulwimi, kunye nokugweba zonke zinokubenza nzima ubomi besigulana kwaye zide zichaphazele isakhono saso sokufuna okanye ukufumana unyango. Ukungakwazi ukunxibelelana neentlungu, iimpawu, okanye ukulandela unyango kusenokubangela ukuba isifo sibe mandundu.
Kumanqanaba okugqibela esi sifo, i-atrophy yengqondo kunye notshintsho lweselula lunokuba nefuthe ekusebenzeni kwesiqhelo. Isigulana sinokuphulukana namandla okulawula intshukumo yamathumbu kunye nesinyi, kwaye sinokuba nobunzima ngokuginya. Ezinye iingxaki zibandakanya izifo ezosulelayo, ukwanda kweziganeko zokuwa, ukungondleki, ukuphelelwa ngamanzi emzimbeni kunye nokutshintsha kwamathumbu.

Ngaba i-Alzheimer's ingathintelwa?

Ngelishwa, ubungqina obukhoyo ngoku bucebisa ukuba ukuthintela isifo sika-Alzheimer akunakwenzeka. Kodwa, ukunqanda ubungozi obunxulunyaniswa ne-Alzheimer's kunokuba luncedo ukuguqula ikhosi yesifo kunye nokunciphisa amathuba okuba nesifo se-Alzheimer ngokukhula kweminyaka. Ngokuziqhelanisa nendlela yokuphila esempilweni njengokuzilolonga mihla le, ukutya ukutya okuneemifuno neziqhamo, ukujonga impilo rhoqo, ukugcina uxinzelelo lwegazi kunye namanqanaba e-cholesterol kulawulo, ukunqanda izinto ezinokuzonwabisa ezinje ngotywala okanye icuba kunokunceda ukugcina imemori kunye nokusebenza kwengqondo kamva ebomini. Ngapha koko, ukuthatha inxaxheba kwimisebenzi efuna ukuqiqa kunye nokuthatha inxaxheba kwimisebenzi ephezulu yengqondo enjengokudlala i-chess, ukusombulula iingxaki kwimathematics, okanye ukudlala imidlalo enzima kunokunceda ekugcineni imisebenzi yengqondo ngokukhula kweminyaka.

Unyango lwezifo ze-Alzheimer

Iziyobisi ezisetyenziswayo ngoku ukunyanga uncedo lwe-Alzheimer ngeempawu. Abayiguquli ikhosi yesifo okanye bayinyange imeko. Ngokuyintloko, zimbini iintlobo zamayeza amiselweyo ngoku kwi-Alzheimer's.

I-Cholinesterase inhibitors

Kwisifo i-Alzheimer's, kukho ukuphelelwa yi-acetylcholine, eyi-neurotransmitter, ethe yanyanzeliswa kwisifundo sesi sifo. Ke ngoko, ukuthintela ii-enzymes ezonakalisa i-acetylcholine kunokuba luncedo kunyango lwe-Alzheimer's.
I-Cholinesterase inhibitors inyusa amanqanaba e-neurotransmitter, i-Acetylcholine ngokuthintela ukonakala kwayo. Zizilwanyana zokuqala ezikhethiweyo kuzo zonke izigulane ezithe zafumanisa ukuba zinesifo se-Alzheimer kwaye ziyakwazi ukuphucula iimpawu. I-cholinesterase inhibitors eziqhelekileyo ezisetyenziselwa ukunyanga isifo se-Alzheimer yi-galantamine, i-rivastigmine, kunye ne-donepezil.

Umchasi we-NMDA receptor antagonist

I-Memantine, i-NMDA receptor antagonist ikwasetyenziswa kunyango lwe-Alzheimer's. Isetyenziswe ngokukodwa kwezi zigulana ezingakwaziyo ukunyamezela unyango lwe-Cholinesterase inhibitors. Kukho ukuphucuka okumodareyitha kweempawu xa unyangwa nge-memantine. Ngelixa unyango oludibeneyo lwe-memantine kunye nezinye i-cholinesterase inhibitors lungakhange lubonakaliswe luluncedo, izifundo ziqhutywa ukujonga naziphi na izibonelelo ezinokubakho.

Elinye iyeza

Iivithamini ezininzi, izongezo, kunye namayeza asetyenziswa kwizigulana ezinesifo se-Alzheimer njengoko kunokuba luncedo ekuphuculeni ukusebenza kwengqondo. Uphononongo oluvavanya izibonelelo zala mayeza alukaqiniseki. Olunye unyango olunokuthi lube neziphumo ezilungileyo zezi:

I-9-Me-BC Powder

I-9-ME-β-Carbolines yimixube ye-pyridoindole, efunyanwe kwiindlela ezingapheliyo nezingaphandle. Uphando lwe-9-ME-β-Carbolines lufumanise ukuba ezi zinto zinokubangela iziphumo ezinje nge-neuroprotection, neurostimulation, anti-inflammatory action, kunye neuroregeneration. Ngaphaya koko, i-9-ME-BC inqanda ukwanda kwee-dopaminergic neurons ngaphandle kokuchaphazela ukuthathwa kwe-dopamine. I-9-ME-BC ibonakalise isenzo sokulwa nokusasazeka kunye neziphumo ezinobungozi obuncinci kwii-neurons.
Iintshukumo ze-9-ME-BC zixolelaniswe nomthuthi we-organic cation, kwaye zikwakhokelela ekubonakalisweni kwemfuza enoxanduva lokudityaniswa kwezinto ezininzi ezibalulekileyo ze-neurotrophic kubandakanya iBDNF, NCAM1, kunye ne-TGFB2. Ezi zinto ze-neurotrophic zibalulekile ekuphumeni kwee-neurites, ezinokuba neenzuzo ze-neurodegenerative kunye ne-neuroprotective xa ii-neurons zihlangana neetyhefu ezahlukeneyo. Yiyo loo nto i-9-ME-BC inezibonelelo ezininzi kwii-neurons ezenza ukuba ibe sisongezelelo esiluncedo ngokuchasene nokuphazamiseka kwemithambozo njengesifo sikaParkinson kunye nesifo se-Alzheimer's.

Umgubo weCMS121

I-CMS121 evela kwi-fisetin yi-neuroprotective compound elawulwa ngomlomo. I-Fisetin sisixhobo se-flavonoid esivela kwiziqhamo kunye nemifuno. Izifundo zibonise ukuba i-fisetin ineziphumo eziluncedo kukuqonda kunye nonxibelelwano lwe-neuronal. Kunye neepropathi zayo ze-antioxidant, i-fisetin inokuphucula amanqanaba ezinto ezikhuselayo kwi-Nervous System. Ngapha koko, i-fisetin nayo ineepropathi ezichasene nokudumba. Zonke ezi zibonelelo ze-fisetin zibonisa ukuba kunokuba luncedo kunyango lwezifo ezinokuphazamiseka kunxibelelwano lwe-neuronal kunye nokusebenza.
Isiphumo se-fisetin, i-CMS121 powder inamaxesha angama-400 aphezulu kunefisetin. I-CMS121 ikwabonakalise iipropathi ezongezelelweyo ezinje ngokuphuculwa kweprofayili ye-pharmacological kunye nozinzo kwifom yayo ebonakalayo ngokufumaneka kakuhle komlomo. I-CMS121 inokuthi ibe sisongezelelo esiluncedo kwizigulana ezinengxaki yokuphazamiseka kwemithambo-luvo njengesifo se-Alzheimer's.

I-CAD31 powder

I-CAD31 ineempembelelo ezininzi eziluncedo ezinokusebenza ekunciphiseni ukonakala okunxulumene nobudala beeuron. Kubonakalisiwe ukukhuthaza iiseli ezinamagama ezivela kwimibungu yabantu ukuze iphinde iphinde iphinde iphinde iphinde yenzeke. Uvavanyo lokuvavanya izibonelelo ze-CAD31 kwimeko yeklinikhi zenziwa kwizifundo zezilwanyana. Iimodeli zeempuku ezinesifo se-Alzheimer zenziwa nge-CAD31. Isifundo siqwalasele ukuphuculwa kwemisebenzi yememori kunye nokwehla kokudumba kwiimodeli zeempuku. Iqukumbele ukuba i-CAD31 inokuba yinkqubo ekhusela i-neuroprotective kwaye iyakwazi ukuwela isithintelo segazi nengqondo ngokufanelekileyo.
I-CAD 31 ikakhulu isebenza ngokwenziwa kwee-synapses kunye neethagethi zendlela ye-metabolic enje ngemetabolism ye-fatty acids. Ezi zifundo zokuqala zineziphumo ezithembisayo zokusetyenziswa kwe-CAD-21 kwiingxaki zemithambo-luvo kubandakanya isifo se-Alzheimer kunye nezinye iintlobo zesifo sengqondo esixhalabisayo.

J147 powder

I-J147 powder ivela kwiCurcumin, yona ngokwayo ivela kwi-spice eyaziwayo yaseNdiya eyaziwa ngokuba yi-turmeric. I-Curcumin yindawo eneziphumo ezaziwayo ezinjengezinto ezichasayo, i-antioxidant, ukunciphisa ityhefu ebangelwa yiprotein, njalo njalo. Ngelishwa, i-curcumin ngokwayo yayingeyiyo eyoncedo olusebenzayo njengoko ine-bioavailability ehlwempuzekileyo kwaye ayinakho ukuwela isithintelo segazi-lobuchopho.
Ngokungafani ne-curcumin, i-J147 powder ineprofayili ezinzileyo ye-pharmacological, ukungena kakuhle kwe-CNS, kunye nokuba ne-bioavailability yomlomo. I-molecule ye-J147 nayo inamaxesha angaphezu kwama-10 aphezulu xa kuthelekiswa ne-curcumin. Izifundo zezilwanyana eziqhutywe ukuza kuthi ga ngoku kwi-J147 powder ibonakalisile ukuba kunokuba luncedo kakhulu kubantu abalupheleyo nakwabo banesifo se-Alzheimer's.

I-Monosialotetrahexosyl ganglioside Sodium (GM1) umgubo

I-Monosialotetrahexosylganglioside sodium (GM1) yikhompawundi eyandayo ethandwayo esetyenziselwa ukunyanga ukuphazamiseka kwemithambo-luvo. Oku ikakhulu ngenxa yesenzo sayo se-neuroprotective. Kodwa ikwanazo nezenzo zokuzikhusela kwimithambo yegazi ebonelela nge-CNS. Kwisifundo esenziwe kwikhompawundi ye-GM1, i-GM1 yafunyanwa inezenzo ezikhuselayo kwii-radicals zasimahla ezenzakalisa ukwenzakala kweseli.
I-neuroprotective, kunye neepropathi zokulwa ne-antioxidant ye-Monosialotetrahexosyl ganglioside Sodium (GM1) powder, yenza ukuba ibe sisongezelelo esinokuba luncedo kwiziphazamiso ezininzi zenkqubo ye-nervous system kubandakanya kodwa kungaphelelanga kwisifo se-Alzheimer's, isifo sika-Parkinson, isifo sengqondo esixhalabisayo, njalo njalo.

I-Octacosanol powder

I-Octacosanol sisixhobo semichiza esivela kwizityalo ezinjengeoyile yentsholongwane yengqolowa kunye neswekile. Ngokolwakhiwo nangokwemichiza, ineempawu ezifanayo neVitamin E. Izifundo ezininzi ziye zafumanisa ukuba i-octacosanol ine-antioxidant, neuroprotective, kunye ne-anti-inflammatory properties. Isetyenziswa ngokubanzi ziimbaleki kwaye ikwasetyenziswa njengesixhobo sokunyanga ukuphazamiseka kwemithambo-luvo njengesifo sika-Parkinson, isifo sika-Alzheimer, isifo sikaLou Gehrig, nezinye ezininzi.

Izifundo eziqhubekayo kwizifo ze-Alzheimer's

Akukho lunyango lwesifo se-Alzheimer okwangoku, kwaye onke amachiza asetyenziswayo kunyango lwe-Alzheimer anokuphucula kuphela iimpawu okwethutyana ngokwandisa isenzo se-neurotransmitters ngaphakathi kwenkqubo ye-nervous system. Kodwa la machiza akanakukuthintela ukuqhubela phambili kwesifo.
Izifundo ezininzi zenziwa ukuqonda ngcono isifo esisisiseko se-etiology kunye ne-pathophysiology yokuphuhlisa unyango ekujolise kulo kwi-Alzheimer's. Abaphandi kule nkalo banethemba lokufumana iindlela zonyango ezinokulibazisa okanye ezinokumisa ukuqhubela phambili kwesifo kwinqanaba eliphambili. Kusenokwenzeka ukuba iindlela zonyango ezizayo azizukubandakanya ichiza elinye, kodwa indibaniselwano yamachiza aliqela asebenza kwiindlela ezininzi.

I-Prognosis ye-Alzheimer's Disease

Ngelixa amayeza aliqela asetyenziselwa ukunyanga isifo se-Alzheimer, anokucutha ukuqhubela phambili kwesifo. Nangona kunjalo, la mayeza axabiseke kakhulu njengoko ephucula amandla okuzimela nokuzenzela imisebenzi yabo yemihla ngemihla ngoncedo oluncinci. Iinkonzo ezahlukeneyo ziyafumaneka ezinikezela ngononophelo kwizigulana ezinesifo i-Alzheimer's. Ngelishwa, akukho nyango lwaziwayo lwe-Alzheimer's.

Imbekiselo:

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